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Research Article

PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin

  • Derek P. Narendra,

    Affiliation: Biochemistry Section, Surgical Neurology Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America

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  • Seok Min Jin,

    Affiliation: Biochemistry Section, Surgical Neurology Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America

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  • Atsushi Tanaka,

    Affiliation: Biochemistry Section, Surgical Neurology Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America

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  • Der-Fen Suen,

    Affiliation: Biochemistry Section, Surgical Neurology Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America

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  • Clement A. Gautier,

    Affiliation: Center for Neurologic Diseases, Brigham and Women's Hospital, Program in Neuroscience, Harvard Medical School, Boston, Massachusetts, United States of America

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  • Jie Shen,

    Affiliation: Center for Neurologic Diseases, Brigham and Women's Hospital, Program in Neuroscience, Harvard Medical School, Boston, Massachusetts, United States of America

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  • Mark R. Cookson,

    Affiliation: Cell Biology and Gene Expression Unit, Laboratory of Neurogenetics, National Institute on Aging, Bethesda, Maryland, United States of America

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  • Richard J. Youle mail

    youler@ninds.nih.gov

    Affiliation: Biochemistry Section, Surgical Neurology Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, United States of America

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  • Published: January 26, 2010
  • DOI: 10.1371/journal.pbio.1000298

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Linked Articles

The Roles of PINK1 and Parkin in Parkinson's Disease

Rachel Jones
Synopsis | published 26 Jan 2010 | PLOS Biology
doi:10.1371/journal.pbio.1000299